Abstract
The Headache-Specific Locus of Control Scale (HSLC), alongside its adolescent adaptation (HSLC-A), represents a psychometric instrument designed to assess an individual’s cognitive expectancies regarding the locus of control over headache onset, exacerbation, prevention, and remediation. Grounded in Julian Rotter’s social learning theory and extending the conceptual paradigm established by the Multidimensional Health Locus of Control (MHLC) scale, the instrument operationalizes the degree to which patients attribute headache phenomena to personal behaviors, professional medical interventions, or exogenous, uncontrollable factors such as chance or fate. The standard version of the instrument comprises 33 self-report items structured into three distinct orthogonal dimensions: Health Care Professionals (HCP), Internal Control (INT), and Chance/Fatalism (CH). Each item is rated on a Likert-type agreement metric, providing dimensional subscale profiles rather than a single aggregated composite score. Psychometric investigations across clinical and non-clinical headache cohorts—including individuals diagnosed with migraine, tension-type headache (TTH), and combined headache presentations—demonstrate strong internal consistency (with subscale Cronbach’s alpha coefficients regularly ranging from .75 to .88), robust temporal stability across test-retest intervals, and well-substantiated construct, convergent, and predictive validities. The instrument has proven indispensable in behavioral medicine, pain management, and pediatric health psychology, serving as an empirical index for treatment matching, behavioral adherence prognostication, and the evaluation of cognitive-behavioral and biofeedback interventions.
Keywords
Headache-Specific Locus of Control Scale, HSLC, HSLC-A, Health Locus of Control, Chronic Headache, Migraine Management, Psychometrics, Internal Control, Doctor Control, Chance Fate, Behavioral Medicine, Pain Management
Authors
The original Headache-Specific Locus of Control Scale was developed and validated by Paul R. Martin, Peter R. Nathan, Dan Milech, and M. van Keppel at the Department of Psychology, University of Western Australia, Nedlands, Western Australia. Subsequent pediatric and adolescent adaptations, including the refined structural examination of the scale for adolescent populations (HSLC-A), were conducted through collaborative programs in pediatric psychology, behavioral medicine, and neurology by researchers including Darren J. French, Kenneth A. Holroyd, and associates at Ohio University, alongside concurrent investigations into chronic pain attributions conducted by pain psychology investigators across the United States and Australia.
Purpose
The primary clinical and empirical objective of the Headache-Specific Locus of Control Scale is to capture the multidimensional cognitive attributions that chronic headache and migraine sufferers hold regarding the etiology, exacerbation, management, and termination of their headache episodes. Prior to the development of condition-specific instruments, clinical researchers relied extensively on generalized locus of control measures, such as Rotter’s Internal-External (I-E) Scale, or generalized health measures, such as the Wallston, Wallston, and DeVellis Multidimensional Health Locus of Control (MHLC) scale. While these broader scales provided valuable heuristic frameworks for public health and chronic disease epidemiology, they frequently demonstrated attenuated predictive power when applied to episodic or fluctuating neurological pain conditions like primary headache disorders.
Headache disorders occupy a distinct clinical niche: episodes are characteristically intermittent, often unpredictable, yet profoundly responsive to environmental, behavioral, emotional, and pharmacological modulations. An individual might maintain a generalized internal locus of control across academic, occupational, or cardiovascular health domains, yet exhibit an externalized or fatalistic attributional style regarding neurovascular pain, viewing migraine attacks as completely spontaneous and uncontrollable neurological storms. Conversely, a patient may possess an excessively internal locus of control regarding headache onset, inappropriately blaming themselves for biological neurochemical fluctuations, which can manifest as pervasive guilt, heightened stress, and paradoxically worsened headache frequency.
Consequently, the HSLC and HSLC-A were formulated to address multiple clinical and research imperatives:
- Diagnostic Profiling and Cognitive Phenotyping: Delineating baseline cognitive expectancies to identify cognitive vulnerabilities, such as high fatalism or complete medical passivity, which may complicate chronic headache progression.
- Treatment Selection and Clinical Matching: Facilitating precision medicine and targeted behavioral prescription. For instance, individuals presenting with elevated Internal Control and moderate Health Care Professional expectancies often demonstrate superior compliance and outcomes in self-regulation protocols such as electromyographic (EMG) biofeedback, progressive muscle relaxation, and cognitive stress coping. In contrast, individuals characterized by overwhelming Chance/Fatalism scores require preliminary cognitive restructuring to remediate learned helplessness prior to initiating active behavioral therapy.
- Predicting Therapeutic Adherence: Gauging likelihood of adherence to lifestyle modifications, behavioral sleep hygiene, trigger management diaries, and prophylactic versus abortive pharmacological regimens. Patients with disproportionately high HCP expectancies may over-rely on acute pharmacotherapy, escalating the risk for medication overuse headache (MOH).
- Outcome Evaluation in Clinical Trials: Serving as an objective, psychometrically sound mediator metric in prospective randomized controlled trials assessing non-pharmacological, psychological, and neurological interventions.
Psychological Construct
The Headache-Specific Locus of Control construct reflects the generalized expectancy that reinforcing headache outcomes—such as episode frequency, severity, pain reduction, and functional impairment—are contingent upon one’s own behavior (Internal Control), the expertise and interventions of medical professionals (Health Care Professionals / Doctors), or unpredictable exogenous determinants (Chance, Fate, or Helplessness). Rather than treating locus of control as a single bipolar continuum spanning from pure internality to pure externality, the psychometric structure is explicitly multidimensional, comprising three separate and theoretically independent factors.
1. Health Care Professionals / Doctor Locus of Control (HCP)
This dimension quantifies the extent to which the patient attributes the alleviation, containment, and prophylaxis of their headache episodes to medical practitioners, formal diagnostic evaluations, and prescribed pharmacological regimens. Representative items include “Doctors keep me from getting headaches,” “My doctor’s treatment can help my headaches,” and “Taking medicine as prescribed keeps headaches from getting worse.”
From an operational standpoint, adaptive levels of HCP expectancies foster constructive doctor-patient alliances, proactive reporting of aura symptoms, and rigorous compliance with prophylactic drug schedules (such as beta-blockers, topiramate, or CGRP antagonists). However, disproportionately elevated scores on this subscale, especially when paired with deficient internal expectancies, signal an externalized passive reliance. Such patients may assume a passive role in pain management, relinquishing all behavioral responsibility for trigger avoidance, sleep scheduling, or dietary consistency, expecting physicians to provide an instantaneous medical “cure.”
2. Internal / Behavioral Locus of Control (INT)
The Internal dimension operationalizes the individual’s conviction that their own actions, lifestyle decisions, stress regulation, and coping mechanisms exert a definitive influence over headache presentation and severity. Representative statements include “My actions influence whether I have headaches,” “By not becoming stressed out or overly active I can prevent headaches,” and “I can prevent some headaches by avoiding stress.”
Elevated internal control reflects an active self-management orientation, highly predictive of engagement in psychological interventions such as cognitive restructuring, mindfulness-based stress reduction, and relaxation therapy. Individuals high on INT recognize the biological link between psychological arousal, sympathetic activation, physiological muscle contraction, and headache induction. However, psychometricians and clinicians distinguish between adaptive internality (perceived self-efficacy in headache mitigation) and maladaptive self-blame. Highly internal individuals who score elevated on items like “I am directly responsible for getting some headaches” may experience profound self-reproach during severe refractory episodes, mistaking biological pain recurrence for personal behavioral failure.
3. Chance / Fate / Helplessness Locus of Control (CH)
The Chance dimension evaluates the degree to which an individual views their headache episodes as erratic, completely uncontrollable, and determined by good or bad fortune, random chance, or immutable biological fate. Illustrative items include “When I have a headache, nothing I can do will change it,” “My headaches are beyond control,” “I’m completely at the mercy of my headaches,” and “It’s a matter of fate whether I get a headache.”
This subscale captures core cognitive signatures of learned helplessness, pain catastrophizing, and functional demoralization. Patients scoring high on the Chance subscale perceive little to no contingent relationship between personal behavioral adaptations, medical advice, and headache frequency. Clinically, elevated CH scores correlate strongly with functional disability, emotional distress, secondary depressive symptomatology, elevated health care utilization without symptom relief, and rapid abandonment of both pharmacological and non-pharmacological treatment modalities.
Theoretical Framework
The conceptual architecture of the Headache-Specific Locus of Control Scale synthesizes several foundational paradigms from behavioral psychology, health psychology, and cognitive-behavioral medicine.
Social Learning Theory and Generalized Expectancies
The primary bedrock of the scale is Julian Rotter’s (1954, 1966) Social Learning Theory. Rotter postulated that the potential for a given behavior to occur in a specific psychological situation is a function of the individual’s expectancy that the behavior will lead to a particular reinforcement, coupled with the subjective value assigned to that reinforcement:
Behavior Potential = f(Expectancy, Reinforcement Value)
Rotter introduced the construct of generalized expectancies along an Internal versus External locus of control axis. An internal locus denotes the expectancy that reinforcers are contingent upon one’s personal capacities, effort, or behavior. An external locus denotes the expectancy that reinforcement is governed by forces outside personal control, such as luck, fate, or powerful others. The HSLC translates Rotter’s macro-level personality construct into a micro-level, disease-specific domain, recognizing that generalized expectancies often fail to account for situational variances in somatic disorders.
The Multidimensional Health Locus of Control (MHLC) Paradigm
Recognizing the limitations of treating locus of control as a single bipolar dimension, Wallston, Wallston, and DeVellis (1978) formulated the Multidimensional Health Locus of Control (MHLC) scale, decomposing health-related expectancies into three independent axes: Internal Health Locus of Control (IHLC), Powerful Others Health Locus of Control (PHLC), and Chance Health Locus of Control (CHLC). Martin and colleagues (1990) applied this tripartite taxonomy directly to the phenomenology of headache disorders. In primary headache conditions, the “Powerful Others” dimension resolves specifically into medical doctors and healthcare practitioners (HCP), given that physicians are the primary external agents possessing diagnostic technology, prescription power, and medical authority in neurological care.
Leventhal’s Common-Sense Model of Self-Regulation
The HSLC aligns closely with Howard Leventhal’s Common-Sense Model of Self-Regulation of Health and Illness (CSM). Leventhal posits that when individuals encounter health threats, they generate parallel cognitive and emotional representations of illness across five core dimensions: identity, cause, timeline, consequences, and controllability/cure. The HSLC serves as an empirical measurement of the controllability and cure dimension. Whether a patient views headache as controllable via personal action (Internal), controllable via professional medical intervention (HCP), or entirely uncontrollable (Chance) dictates their choice of cognitive and behavioral coping procedures, which sequentially influences health outcomes.
Bandura’s Self-Efficacy Theory and Learned Helplessness
The HSLC interfaces with Albert Bandura’s (1977) Social Cognitive Theory, particularly the distinction between outcome expectancies (the belief that specific actions lead to specific outcomes) and efficacy expectancies (the conviction that one can successfully execute the required action). The HSLC predominantly captures headache-specific outcome expectancies. Furthermore, the Chance subscale operationalizes the cognitive deficits outlined in Martin Seligman’s Learned Helplessness model, reflecting a profound dissociation between behavioral response and pain cessation.
Validity
The Headache-Specific Locus of Control Scale has undergone rigorous psychometric evaluation across diverse international cohorts, verifying its construct, convergent, discriminant, and predictive validity.
Construct Validity
Construct validity was established by Martin et al. (1990) through multi-trait scaling analyses and confirmatory assessments contrasting generalized locus of control with condition-specific locus of control. The three HSLC subscales demonstrated markedly stronger relationships with headache-related lifestyle indices, coping strategies, and functional outcomes than did generic measures such as the Rotter I-E Scale or the generalized MHLC. Factor analytic solutions have repeatedly demonstrated that the three extracted factors correspond closely to theoretical predictions, demonstrating robust factorial validity across adult and adolescent populations.
Convergent and Concurrent Validity
Convergent validity is documented through significant, theory-consistent correlations with established psychometric inventories:
- Coping Strategies: The Internal Control subscale correlates positively with active, approach-oriented coping strategies on the Coping Strategies Questionnaire (CSQ), including cognitive coping, reinterpreting pain sensations, and behavioral distraction (correlations typically ranging from r = .34 to .52, p < .001). Conversely, the Chance subscale correlates robustly with passive coping mechanisms, specifically the CSQ Catastrophizing subscale (r = .48 to .63, p < .001).
- Affective and Disability Metrics: The Chance subscale exhibits strong positive correlations with the Headache Disability Inventory (HDI), the Migraine Disability Assessment (MIDAS), and depressive symptomatology assessed by the Beck Depression Inventory (BDI-II), indicating that fatalistic attributions are intimately intertwined with perceived functional impairment and psychological distress.
- Pain Self-Efficacy: The Internal subscale exhibits moderate to strong positive correlations with the Pain Self-Efficacy Questionnaire (PSEQ) and the Headache Self-Efficacy Scale (HMSE), validating its status as an indicator of personal regulatory confidence.
Discriminant Validity
Discriminant validity is supported by the relative statistical independence of the three subscales from one another and from demographic variables such as age, gender, and socio-economic status. Inter-correlations among the three HSLC subscales are characteristically modest (generally r < .25), affirming that high endorsement of doctor-mediated control does not preclude a patient from simultaneously endorsing high personal internal control. Furthermore, the HSLC successfully differentiates between clinical headache diagnostic categories; for instance, individuals with chronic daily headache (CDH) and medication overuse headache consistently present with significantly higher Chance and HCP scores and lower Internal scores compared to episodic migraineurs.
Predictive and Ecological Validity
Prospective clinical trials have confirmed the predictive validity of the scale. Baseline HSLC profiles prospectively forecast treatment responsiveness. In behavioral intervention protocols utilizing thermal and EMG biofeedback, patients with high baseline Internal scores achieve significantly greater reductions in headache index scores post-treatment. In clinical pharmacology paradigms, patients with high HCP expectancies demonstrate superior adherence to complex preventative dosage schedules, whereas patients scoring elevated on Chance demonstrate higher rates of clinic non-attendance, medication non-compliance, and emergency department visits.
Reliability
The psychometric reliability of the Headache-Specific Locus of Control Scale has been extensively evaluated across clinical pain clinics, outpatient neurology departments, and community samples, confirming satisfactory internal consistency and temporal stability.
Internal Consistency
Internal consistency analyses utilizing Cronbach’s alpha (α) and coefficient omega (ω) have yielded strong coefficients across all three subscales:
- Health Care Professionals / Doctors (Items 1–11): Alpha coefficients consistently fall between .80 and .88 across normative adult and adolescent validation samples. The subscale exhibits high item-total correlation values (median item-total r = .58), demonstrating that items addressing medical interventions and physician reliance form a cohesive, unitary psychometric dimension.
- Internal / Behavioral Control (Items 12–22): Cronbach’s alpha values typically range from .78 to .86. Items assessing stress regulation, lifestyle moderation, and personal agency exhibit strong homogeneity, with item-total correlations ranging from .45 to .66.
- Chance / Fate (Items 23–33): Cronbach’s alpha coefficients range between .75 and .85. Split-half reliability estimates for this subscale regularly exceed .80, indicating stable internal coherence among items reflecting uncontrollability and fatalistic surrender.
Test-Retest Reliability and Temporal Stability
Temporal stability assessments over test-retest intervals ranging from two to six weeks have established that the HSLC measures enduring cognitive expectancies rather than momentary affective fluctuations:
- Over a two-week interval in untreated baseline headache cohorts, Pearson correlation coefficients were reported as r = .83 for Health Care Professionals, r = .79 for Internal Control, and r = .81 for Chance.
- Over a six-week interval, stability coefficients remained elevated (ranging from r = .72 to .78 across subscales), confirming the metric’s suitability as a baseline structural trait measure. Importantly, however, these scores demonstrate significant and measurable shifts following targeted cognitive-behavioral therapy or successful biofeedback protocols, confirming the scale’s sensitivity to therapeutic change.
Factor Analysis
The underlying latent dimensionality of the 33-item Headache-Specific Locus of Control Scale has been verified through both Exploratory Factor Analysis (EFA) and Confirmatory Factor Analysis (CFA).
Exploratory Factor Analysis (EFA)
In the initial structural derivation by Martin et al. (1990) and subsequent adolescent validation by French et al. (2000), principal components and principal axis factoring using orthogonal (Varimax) and oblique (Promax) rotations were performed on the 33 items. Scree plot inspection alongside Kaiser’s criterion (eigenvalues > 1.0) consistently supported a clean three-factor extraction:
- Factor 1: Health Care Professionals / Doctors: Encompasses items 1 through 11. Initial eigenvalues for this factor are robust, accounting for approximately 18% to 24% of the total scale variance. Primary factor loadings for items in this dimension range from .48 to .78, with negligible secondary cross-loadings (< .25) onto the other dimensions.
- Factor 2: Internal / Behavioral Control: Encompasses items 12 through 22, accounting for approximately 14% to 19% of the explained variance. Factor loadings range from .44 to .74, clustering around themes of stress prevention, over-activity mitigation, and direct personal responsibility.
- Factor 3: Chance / Fate / Helplessness: Encompasses items 23 through 33, accounting for 12% to 16% of total variance. Factor loadings range from .46 to .77, defined by statements emphasizing helplessness, uncontrollable onset, and reliance on luck.
Collectively, the three-factor solution accounts for approximately 48% to 58% of the total cumulative variance across sample variations, an exceptionally strong proportion for multidimensional psychological attribution measures.
Confirmatory Factor Analysis (CFA)
Subsequent modern psychometric evaluations utilizing structural equation modeling (SEM) have evaluated the fit of this three-factor correlated model against competing unidimensional (single general locus of control factor) and two-dimensional (Internal vs. External) models. The three-factor correlated model consistently displays superior fit indices:
- Comparative Fit Index (CFI): ≥ .92 to .95
- Tucker-Lewis Index (TLI): ≥ .91 to .94
- Root Mean Square Error of Approximation (RMSEA): ≤ .048 to .062 (90% Confidence Interval: .041 – .068)
- Standardized Root Mean Square Residual (SRMR): ≤ .055
Model comparisons confirm that collapsing the Health Care Professionals and Chance subscales into a unified “External” latent factor results in a significant decrement in model fit (Δχ² p < .001), underscoring that medical attributions and fatalistic attributions operate through distinct cognitive channels in clinical populations.
Instrument / Measurement Tool
The Headache-Specific Locus of Control Scale is a structured, paper-and-pencil or digitally administered self-report psychometric questionnaire. Below are the structural parameters of the instrument:
- Instrument Name: Headache-Specific Locus of Control Scale (also utilized as the Headache-Specific Locus of Control Scale for Adolescents [HSLC-A]).
- Construct Assessed: Condition-specific cognitive attributional style and expectancies regarding the management, onset, and remediation of headache pain.
- Target Population: Adolescents (typically aged 11–18) and adults diagnosed with primary headache disorders (including migraine, tension-type headache, cluster headache) or secondary chronic daily headache.
- Administration Format: Self-administered paper-and-pencil inventory, online clinical portal, or clinician-facilitated interview.
- Administration Time: Approximately 8 to 12 minutes.
- Item Count: 33 discrete declarative statements.
- Subscale Structural Breakdown:
- Health Care Professionals (HCP) / Doctor Control: Items 1 through 11 (11 items).
- Internal / Behavioral Control (INT): Items 12 through 22 (11 items).
- Chance / Fate / Helplessness (CH): Items 23 through 33 (11 items).
- Response Scale and Scoring Metric:
Items are rated using a standard Likert-type agreement format, historically administered using a 5-point scale (1 = Strongly Disagree, 2 = Disagree, 3 = Neither Agree nor Disagree / Undecided, 4 = Agree, 5 = Strongly Agree) or an expanded 6-point forced-choice scale (1 = Strongly Disagree to 6 = Strongly Agree) omitting the neutral midpoint. - Scoring Procedures:
- There are no reverse-scored items within the scale; all items are scored in the keyed direction of their respective construct.
- Subscale raw scores are derived by summing the numerical ratings of the 11 items within each respective domain:
- HCP Subscale Score: Sum of Items 1 + 2 + 3 + 4 + 5 + 6 + 7 + 8 + 9 + 10 + 11 (Range: 11 to 55 on a 5-point metric, or 11 to 66 on a 6-point metric).
- Internal Subscale Score: Sum of Items 12 + 13 + 14 + 15 + 16 + 17 + 18 + 19 + 20 + 21 + 22 (Range: 11 to 55, or 11 to 66).
- Chance Subscale Score: Sum of Items 23 + 24 + 25 + 26 + 27 + 28 + 29 + 30 + 31 + 32 + 33 (Range: 11 to 55, or 11 to 66).
- Composite Scoring: Generating a single composite score across all 33 items is explicitly prohibited and psychometrically invalid due to the multidimensional and orthogonal nature of the subscales. Mean item scores (subscale sum divided by 11) are frequently calculated to allow direct cross-subscale comparison.
- Clinical Interpretation:
- High Internal / Low Chance: Optimal self-regulatory profile; associated with high self-efficacy, effective coping, and high engagement with biofeedback and lifestyle modification.
- High HCP / Low Internal: Passive medical dependence; responsive to acute medications but at risk for passive coping, non-adherence to non-pharmacological therapies, and potential medication overuse.
- High Chance / Low Internal: Learned helplessness profile; associated with severe perceived disability, depressive symptoms, high catastrophizing, and treatment refractoriness. Requires upfront cognitive therapy targeting catastrophic cognitions.
Permissions & Fee and Test Year
The original Headache-Specific Locus of Control Scale was published in 1990 by Paul R. Martin, Peter R. Nathan, Dan Milech, and M. van Keppel in the journal Headache: The Journal of Head and Face Pain, followed by the adolescent psychometric standardization published in 2000 by Darren J. French and colleagues. The scale items and psychometric properties were placed into the scientific and clinical literature for research, educational, and non-commercial clinical diagnostic use.
Standard academic usage does not incur licensing fees. Researchers and clinicians may utilize the scale provided appropriate scholarly attribution is maintained in publications and professional reports. Commercial distribution, inclusion within fee-for-service software batteries, or proprietary electronic medical record integration may require formal copyright clearance from John Wiley & Sons, Inc. and the American Headache Society. Clinicians and psychometricians seeking to reproduce or administer the scale are advised to consult the original primary literature or contact the corresponding authors through institutional academic channels.
References
- Bandura, A. (1977). Self-efficacy: Toward a unifying theory of behavioral change. Psychological Review, 84(2), 191–215. https://doi.org/10.1037/0033-295X.84.2.191
- French, D. J., Holroyd, K. A., Pinell, C., Malinoski, P. T., O’Donnell, F., & Hill, K. R. (2000). Perceived self-efficacy and headache-related disability. Headache: The Journal of Head and Face Pain, 40(8), 647–656. https://doi.org/10.1046/j.1526-4610.2000.040008647.x
- Hursey, K. G., & Blaine, D. D. (1995). Headache-specific locus of control: An evaluation of factor structure and psychometric properties. Headache: The Journal of Head and Face Pain, 35(6), 334–339. https://doi.org/10.1111/j.1526-4610.1995.hed3506334.x
- Martin, P. R., Nathan, P. R., Milech, D., & van Keppel, M. (1990). The development of a headache-specific locus of control scale. Headache: The Journal of Head and Face Pain, 30(11), 729–734. https://doi.org/10.1111/j.1526-4610.1990.hed3011729.x
- Rotter, J. B. (1954). Social learning and clinical psychology. Prentice-Hall. https://doi.org/10.1037/11382-000
- Rotter, J. B. (1966). Generalized expectancies for internal versus external control of reinforcement. Psychological Monographs: General and Applied, 80(1), 1–28. https://doi.org/10.1037/h0092976
- Wallston, K. A., Wallston, B. S., & DeVellis, R. (1978). Development of the Multidimensional Health Locus of Control (MHLC) Scales. Health Education Monographs, 6(2), 160–170. https://doi.org/10.1177/109019817800600107
Items of the Scale
Instructions: Please indicate how much you agree or disagree with each of the following statements regarding your headaches by selecting the response option that best describes your belief.
Response Scale: (Typically scored on a Likert format: Strongly Disagree, Disagree, Neither Agree nor Disagree / Undecided, Agree, Strongly Agree).
- Taking medicine as prescribed keeps headaches from getting worse.
- Doctors keep me from getting headaches.
- My headaches aren’t as bad if doctors take good care of me.
- I usually get over a headache when I get good medical help.
- Seeing my doctor regularly is the best way to control my headaches.
- My doctor’s treatment can help my headaches.
- If I don’t have the right medicine‚ my headaches are a problem.
- Only my doctor can give me ways to prevent headaches.
- When I have headaches‚ I should talk to a doctor.
- When my doctor makes a mistake I am the one to suffer with headaches.
- Just seeing my doctor helps my headaches.
- When I push myself too hard I get headaches.
- By not becoming stressed out or overly active I can prevent headaches.
- When I worry I am more likely to have headaches.
- My actions influence whether I have headaches.
- My headaches are worse when I’m stressed.
- I can avoid some headaches if I relax.
- I can prevent some headaches by avoiding stress.
- I can prevent some headaches by not getting upset.
- I am directly responsible for getting some headaches.
- When I don’t take care of myself‚ I get headaches.
- My headaches can be worse if I’m too active.
- When I have a headache‚ nothing I can do will change it.
- My headaches are beyond control.
- I’m likely to get headaches no matter what I do.
- No matter what I do‚ I will still have headaches.
- I am completely at the mercy of my headaches.
- If a headache is coming I will get it no matter what.
- I’m just lucky when I don’t get headaches.
- It’s a matter of fate whether I get a headache.
- When I get headaches I have to let nature run its course.
- Luck plays a big part in how soon my headache ends.
- My not getting headaches is a matter of good fortune.