Auditory Pathway: Neural Highway of Sound

The auditory pathway represents one of the most intricate and metabolically active sensory networks in the mammalian central nervous system, transforming mechanical fluctuations of ambient air into rich conscious perceptions of speech, music, and environmental soundscapes. Spanning from peripheral mechanoreceptors within the cochlea to multi-tiered computational centers in the cerebral cortex, this ascending neural continuum preserves fine spectral details while simultaneously calculating microsecond-level temporal disparities to localize sound sources in three-dimensional space.

Auditory Pathway

1. Concise Definition

The auditory pathway is the hierarchical anatomical circuit of peripheral nerves, brainstem nuclei, midbrain relays, thalamic gateways, and cortical areas responsible for transducing acoustic energy into electrical impulses and processing that auditory information. It comprises both an ascending (afferent) conduit that directs sound information upward to the brain and a reciprocal descending (efferent) system that modulates peripheral acoustic sensitivity and selective attention.

At its core, this pathway functions not as a passive transmission cable, but as an active parallel-processing computational system. As acoustic signals propagate along the neuroaxis, specialized subcortical nuclei extract distinct sound features—such as frequency, duration, intensity, phase, and spatial origin—before delivering these synthesized representations to the auditory cortex for perceptual identification, emotional evaluation, and linguistic decoding.

2. Etymology & Linguistic Origin

The term auditory traces its roots to the classical Latin verb audire, meaning “to hear,” which gave rise to the Late Latin adjective auditorius, signifying “pertaining to hearing or listening.” The word entered early modern English in the late sixteenth and early seventeenth centuries to classify anatomical structures dedicated to acoustic sensation, notably appearing in pioneering anatomical tracts describing the nervus auditorius.

The English noun pathway is a Germanic compound formed from Old English pæth (a trodden track, course, or route) and weg (a way, direction, or journey). When coupled in modern neuroanatomy during the late nineteenth and early twentieth centuries, auditory pathway became the standard scientific designation for the tract of myelinated axons and synaptic stations that funnel auditory data from the sensory periphery into the telencephalon.

3. Pronunciation & Grammatical Form

Pronunciation: /ˈɔːdɪtəri ˈpæθweɪ/ (British English: [ˈɔː.dɪ.tə.ɹi ˈpɑːθ.weɪ]; American English: [ˈɑː.də.tɔːr.i ˈpæθ.weɪ]).

Grammatical Form: Compound noun phrase. The singular form is auditory pathway; the plural form is auditory pathways. It functions primarily as a countable substantive in anatomical, neurological, and physiological contexts (e.g., “ascending auditory pathways exhibit a strict tonotopic arrangement”). It also appears attributively in phrases such as “auditory pathway lesions” and “auditory pathway neuroplasticity.”

4. Detailed Conceptual Explanation

Understanding the auditory pathway requires following the conversion of acoustic pressure waves into cognitive auditory objects. The journey begins when sound waves strike the tympanic membrane, setting the middle ear ossicular chain (malleus, incus, and stapes) into mechanical vibration. The stapes footplate drives fluid waves within the perilymph-filled scala vestibuli and scala tympani of the cochlea. These fluid displacements deflect the basilar membrane, shearing the stereocilia of inner hair cells against the tectorial membrane. Mechanical displacement of stereocilia opens mechanically gated ion channels, initiating potassium-driven depolarization, calcium influx, and glutamate exocytosis onto type I spiral ganglion neurons.

The myelinated central axons of these bipolar spiral ganglion neurons coalesce into the cochlear branch of the vestibulocochlear nerve (cranial nerve VIII), penetrating the brainstem at the pontomedullary junction. Here, every afferent fiber bifurcates systematically to synapse in the cochlear nucleus complex, which is divided into the dorsal cochlear nucleus (DCN), anteroventral cochlear nucleus (AVCN), and posteroventral cochlear nucleus (PVCN). Different neuronal cell types within these nuclei (including spherical bushy cells, globular bushy cells, octopus cells, and pyramidal cells) perform parallel feature extraction, preserving phase-locking, enhancing temporal precision, or responding to spectral notches.

From the cochlear nucleus, second-order axons diverge along multiple routes. Projections form the acoustic striae (trapezoid body, intermediate acoustic stria, and dorsal acoustic stria). A critical contingent of axons terminates bilaterally in the superior olivary complex (SOC) within the caudal pons. The SOC is the earliest station in the central nervous system where inputs from both ears converge, making it the primary site for binaural sound localization. Neurons in the medial superior olive (MSO) compute microsecond interaural time differences (ITDs), whereas neurons in the lateral superior olive (LSO) calculate interaural level differences (ILDs) assisted by glycinergic inhibitory interneurons from the medial nucleus of the trapezoid body (MNTB).

Ascending fibers from the superior olive and cochlear nuclei collect into a prominent tract known as the lateral lemniscus, which ascends through the rostral pons and midbrain. Interspersed along this tract are the nuclei of the lateral lemniscus (NLL), which process temporal patterns, monaural cues, and acoustic duration. All ascending auditory streams converge in the inferior colliculus (IC) of the tectum. The central nucleus of the inferior colliculus (CNIC) serves as an indispensable midbrain integration hub, consolidating binaural spatial coordinates, complex modulation patterns, and spectral envelopes into an organized topological map.

From the inferior colliculus, signals travel along the brachium of the inferior colliculus to the medial geniculate body (MGB) of the thalamus. The MGB consists of ventral (MGV), dorsal (MGD), and medial (MGM) subdivisions. The MGV represents the lemniscal, highly tonotopic core that routes frequency-specific data straight to the primary auditory cortex (A1) via the geniculocortical tract (acoustic radiation). The dorsal and medial divisions belong to the non-lemniscal, polysensory systems, projecting to associative auditory areas, limbic circuits, and the amygdala, where auditory inputs trigger emotional, autonomic, and startle responses.

The final destination of the classical ascending pathway is the auditory cortex, seated on the superior temporal plane within the transverse temporal gyri of Heschl (Brodmann areas 41 and 42). The primary auditory cortex preserves the tonotopic organization established at the cochlear basilar membrane. From A1, information flows laterally and circumferentially into belt and parabelt secondary auditory fields, subsequently diverging into dual processing streams: a ventral “what” stream projecting toward the anterior temporal lobe and prefrontal cortex for object and speech identification, and a dorsal “where/how” stream passing through parietal regions to frontal motor areas for spatial localization and sensorimotor integration.

5. Historical Development

The classical understanding of auditory neuroanatomy emerged incrementally through dissections, clinical-pathological correlation, and electrophysiological experimentation across centuries:

• Renaissance to the 18th Century: Early anatomists such as Gabriel Falloppio and Bartolomeo Eustachi meticulously illustrated the middle and inner ear chambers, but the central neural connections of cranial nerve VIII remained largely an undifferentiated white matter mass entering the brainstem.
• The 19th Century and Histological Breakthroughs: In 1851, the Italian anatomist Alfonso Corti discovered the spiral acoustic sensory organ inside the cochlea. Hermann von Helmholtz published Die Lehre von den Tonempfindungen als physiologische Grundlage für die Theorie der Musik in 1863, formulating the “resonance theory” of hearing, which proposed that different fibers of the basilar membrane resonate to distinct acoustic frequencies. Concurrently, pioneering neuroanatomists including Camillo Golgi, Santiago Ramón y Cajal, and Paul Flechsig applied novel metal-impregnation and myelin-staining methodologies to map out the trapezoid body, the cochlear nuclei, and the distinct ascending course of the lateral lemniscus.
• Early to Mid-20th Century: Georg von Békésy performed landmark micromechanical experiments on human and animal cadaveric temporal bones, demonstrating that sound creates a mechanical “traveling wave” along the basilar membrane with frequency-dependent displacement peaks. For this, Békésy received the Nobel Prize in Physiology or Medicine in 1961. In 1948, Lloyd A. Jeffress published his seminal theoretical model explaining how delay lines and coincidence detector cells within the superior olivary complex compute interaural time disparities.
• The Electrophysiological Revolution (1960s–1970s): The invention of microelectrode single-unit recording enabled researchers like Jerzy Rose and Nelson Kiang to verify phase-locking, tuning curves, and tonotopic maps across individual auditory brainstem and cortical units. In 1971, Donald Jewett and John Williston demonstrated that volume-conducted scalp electrodes could record subcortical auditory electrical activity, establishing the modern Auditory Brainstem Response (ABR).
• Late 20th Century to Present: Non-invasive neuroimaging (PET, fMRI, MEG) and diffusion tractography have illuminated human central auditory architecture in vivo, confirming the dual-stream model proposed by Josef Rauschecker and David Poeppel and clarifying structural neuroplasticity after sensory deprivation.

6. Theoretical Foundations

Multiple neurobiological and computational paradigms explain how sound is represented and transformed along the auditory pathway:

The foundational paradigm is Tonotopy (or place coding), established by Helmholtz and mathematically codified by Békésy. Because the basilar membrane is narrow, thick, and stiff at its basal turn (responding optimally to high frequencies) and wide, thin, and compliant at its apical apex (responding to low frequencies), acoustic pitch is mapped spatially. This physical coordinate system is preserved across every central node of the pathway: the spiral ganglion, cochlear nucleus, lateral lemniscus, central nucleus of the inferior colliculus, ventral medial geniculate body, and primary auditory cortex all exhibit precise tonotopic frequency gradients.

Complementing place coding is the Temporal (Volley) Theory, proposed by Ernest Glen Wever and Charles Bray in 1930. For acoustic signals below approximately 4 to 5 kHz, auditory nerve fibers fire action potentials aligned to a specific phase of the acoustic wave cycle—a property termed phase-locking. Although individual neurons cannot fire faster than their refractory limits allow (roughly 1 kHz), populations of fibers alternate firing to match higher cyclic frequencies, providing an accurate temporal metric for pitch and timbre.

Spatial perception relies on the Duplex Theory of sound localization, initially articulated by Lord Rayleigh in 1907. It posits that the central auditory system splits sound localization into two distinct physiological streams based on acoustic wavelength: low-frequency sounds (under 1.5 kHz) generate negligible acoustic shadows around the head, relying instead on interaural time differences (ITDs) analyzed in the MSO; conversely, high-frequency sounds (above 1.5 kHz) are scattered by the head, generating interaural level differences (ILDs) computed within the LSO.

Finally, modern cognitive neuroscience models the pathway through the lens of Hierarchical Predictive Coding. Rather than functioning as a pure feedforward cascade, the central auditory pathway exhibits extensive reciprocal efferent connectivity. Descending corticofugal fibers project backward from the cortex to the thalamus, midbrain, brainstem, and ultimately to outer hair cells via the olivocochlear bundle. Under predictive coding models, higher centers continuously project descending top-down predictions regarding incoming acoustic inputs, while ascending afferents primarily convey residual prediction errors, enabling listeners to isolate relevant signals in noisy acoustic environments.

7. Key Components, Types & Dimensions

The auditory pathway is organized into clear peripheral, subcortical, and cortical compartments, alongside an equally critical descending inhibitory circuit:

• Peripheral Mechanoreceptors: The organ of Corti contains a single row of inner hair cells (the genuine sensory transducers receiving 95% of afferent innervation) and three rows of outer hair cells (electromotile amplifiers driven by the motor protein prestin).
• First-Order Neurons (Cranial Nerve VIII): The spiral ganglion houses approximately 30,000 to 50,000 bipolar neurons in humans whose peripheral axons innervate hair cells and whose central axons form the acoustic nerve.
• Medullary Relay (Cochlear Nuclei): Located at the cerebellopontine angle, the AVCN, PVCN, and DCN sort incoming acoustic data into temporal, transient, and spectral feature channels.
• Pontine Binaural Engine (Superior Olivary Complex): Houses the MSO (temporal ITD computation), LSO (intensity ILD computation), and MNTB (glycinergic interneurons providing crossed inhibition essential for level-difference analysis).
• Ascending Fiber Highway (Lateral Lemniscus & Nuclei): A thick tract running through the brainstem containing intermediate processing stations (DNLL and VNLL) that process temporal patterns and binaural monaural cues.
• Midbrain Convergence Station (Inferior Colliculus): Consists of a central tonotopic nucleus (CNIC), a dorsal cortex, and an external cortex. Integrates acoustic cues with somatosensory inputs, motor reflexes, and acoustic startle circuits.
• Thalamic Gateway (Medial Geniculate Body): Subdivided into the ventral division (strict tonotopic acoustic transmission), medial division (multisensory convergence and arousal), and dorsal division (auditory association and limbic routing).
• Cerebral Cortical Processing Stations: Located on the superior temporal gyrus, incorporating the core (A1; Heschl’s gyrus), the secondary belt fields, the associative parabelt, and downstream language regions like Wernicke’s area.
• The Descending Efferent System: The corticofugal pathways and the olivocochlear bundle (Rasmussen’s bundle), which projects from the superior olive back to the cochlea. Medial olivocochlear (MOC) fibers terminate on outer hair cells to turn down gain and prevent acoustic trauma; lateral olivocochlear (LOC) fibers synapse on inner hair cell afferents to regulate synaptic transmission and prevent excitotoxic injury.

8. Examples & Illustrative Cases

Pathologies disrupting specific segments of the auditory pathway reveal how specialized each level of this circuit is:

Clinical Case 1: Acoustic Neuroma (Vestibular Schwannoma). A 52-year-old patient presents with progressive unilateral high-frequency sensorineural hearing loss, continuous unilateral tinnitus, and poor speech discrimination out of proportion to pure-tone thresholds. Magnetic resonance imaging (MRI) reveals a benign schwannoma arising from the vestibular nerve sheath within the internal auditory canal, physically compressing the adjacent cochlear nerve. This case highlights how compression of cranial nerve VIII desynchronizes action potential propagation, disrupting temporal phase-locking and impairing speech comprehension before complete peripheral deafness occurs.

Clinical Case 2: Auditory Neuropathy Spectrum Disorder (ANSD). A child fails universal newborn hearing screening despite exhibiting normal otoacoustic emissions (OAEs), which confirm intact outer hair cell function. Diagnostic workup reveals a complete absence or severe disruption of the Auditory Brainstem Response (ABR). Genetic analysis detects mutations in the OTOF gene, which encodes otoferlin, a critical protein for synaptic exocytosis at the inner hair cell ribbon synapse. The mechanoreceptors successfully transduce acoustic vibrations into receptor potentials, but the signal cannot reliably cross the chemical synapse to trigger synchronized action potentials in the auditory nerve.

Everyday Phenomenon: The Cocktail Party Problem. In a crowded room filled with competing talkers, a listener can selectively focus their attention on a single conversation while ignoring surrounding babble. This feat requires the superior olivary complex to isolate subtle interaural time and level differences, the inferior colliculus to segregate spatial sound streams, and corticofugal feedback loops to dynamically dampen neural responses to non-attended acoustic streams.

9. Measurement & Assessment

Evaluating the integrity of the auditory pathway requires a battery of objective electrophysiological and behavioral assessments:

• Auditory Brainstem Response (ABR / BAEP): The gold-standard electrophysiological test for brainstem pathway conduction. Scalp surface electrodes record far-field electrical potentials generated within the first 10 milliseconds following a click or tone-burst stimulus. The resulting waveform features five characteristic peaks: Wave I (peripheral auditory nerve), Wave II (central auditory nerve / cochlear nucleus), Wave III (superior olivary complex), Wave IV (lateral lemniscus), and Wave V (inferior colliculus). Prolonged interpeak latencies (e.g., I–III or III–V intervals) pinpoint demyelinating lesions or retrocochlear tumors.
• Otoacoustic Emissions (OAEs): Sensitive microphones placed in the external ear canal record low-level acoustic energy produced by outer hair cells during active cochlear amplification. Normal OAEs accompanied by abnormal ABR waveforms represent the classic diagnostic signature of Auditory Neuropathy Spectrum Disorder.
• Pure-Tone and Speech Audiometry: Behavioral tests that determine the minimum audible sound pressure levels across frequencies (125 Hz to 8000 Hz) alongside speech recognition thresholds (SRT) and word recognition scores (WRS) in quiet and noisy backgrounds.
• Cortical Auditory Evoked Potentials (CAEPs): Longer-latency potentials (such as P1, N1, P2, and the cognitive P300 and Mismatch Negativity [MMN]) that evaluate thalamocortical transmission, acoustic discrimination, and pre-attentive sensory memory.
• Diffusion Tensor Imaging (DTI) & Functional Neuroimaging: Advanced neuroimaging techniques that trace white matter tracts (such as the acoustic radiation and lateral lemniscus) and map blood-oxygen-level-dependent (BOLD) responses in human auditory cortex fields during auditory processing tasks.

10. Applications & Practical Significance

Knowledge of the auditory pathway informs medical diagnostics, neurotechnological interventions, and audio engineering:

In otology and neurosurgery, neuro-monitoring of auditory pathway integrity via continuous intraoperative ABR is crucial during cerebellopontine angle surgery (such as vestibular schwannoma resection or microvascular decompression). Shifts in Wave V latency alert surgeons to mechanical traction or ischemia of the cochlear nerve, helping preserve residual hearing.

In bioengineering, cochlear implants bypass damaged or missing hair cells by threading an electrode array directly into the scala tympani of the cochlea, electrically stimulating spiral ganglion cells along their tonotopic axes. For patients with bilateral destruction of the cochlear nerves (for example, caused by Neurofibromatosis Type 2), Auditory Brainstem Implants (ABIs) place a multi-channel surface electrode array directly on the dorsal cochlear nucleus on the floor of the fourth ventricle, restoring functional sound sensation.

In consumer electronics, spatial computing, and acoustic architecture, understanding the physiological mechanisms of the auditory pathway underpins the creation of 3D audio algorithms. Virtual reality headsets synthesize Head-Related Transfer Functions (HRTFs), simulating the pinna spectral notches, interaural time delays, and sound level discrepancies that our superior olivary complexes and inferior colliculi use to locate objects in space.

11. Research & Empirical Evidence

Empirical research has clarified how early acoustic experience shapes the structural development of the auditory pathway. Studies by Michael Merzenich and colleagues demonstrated profound tonotopic reorganization within the primary auditory cortex following peripheral frequency-specific cochlear lesions or intensive perceptual training. When sensory inputs from a given frequency band are abolished, the cortical territory that previously represented those frequencies is colonized by adjacent intact frequencies.

Research led by Andrej Kral on congenitally deaf animal models (such as deaf white cats) identified a distinct developmental critical period for auditory pathway maturation. Without acoustic stimulation, ascending projections fail to form normal synaptic connections within deep cortical layers of A1. However, if cochlear implantation is performed early in life, cross-modal reorganization (where visual or somatosensory inputs hijack the auditory cortex) is prevented, allowing normal central auditory processing to take shape.

More recently, Nina Kraus and her colleagues at Northwestern University have used the subcortical Auditory Brainstem Response to complex sounds (the speech-evoked Frequency Following Response, or FFR) to evaluate how life experiences alter the brainstem. Their studies show that formal musical training and lifelong bilingualism sharpen subcortical temporal precision and pitch tracking, whereas conditions like dyslexia, autism spectrum disorders, and chronic noise exposure correlate with degraded, noisy brainstem signal transmission.

12. Cultural & Cross-Cultural Considerations

The neurobiology of the auditory pathway interacts directly with linguistic and cultural environments. Cross-cultural neurophysiological studies indicate that native speakers of tonal languages (such as Mandarin Chinese, Cantonese, or Vietnamese) demonstrate higher fidelity pitch tracking along the auditory brainstem pathway than native speakers of non-tonal languages (such as English). Because lexical pitch patterns change word meanings in tonal languages, lifelong linguistic experience tunes the subcortical auditory pathway to track fundamental frequency changes with enhanced precision.

Acoustic and musical traditions also shape auditory processing. Musicians trained in distinct tuning systems (such as Indian classical raga scales or Western equal temperament) show distinct cortical evoked potentials (Mismatch Negativity) when presented with microtonal pitch variations outside their cultural framework. Furthermore, structural access to healthcare affects how auditory pathway disorders are diagnosed and managed worldwide: universal newborn hearing screening using ABR and OAE metrics is ubiquitous in high-income nations, but remains limited across many developing regions, underscoring disparities in early intervention for pediatric hearing disorders.

13. Criticisms, Debates & Limitations

Despite significant progress, several core neurophysiological debates concerning the auditory pathway remain active:

The Jeffress Delay-Line Controversy in Mammalian Hearing: While the Jeffress model—which relies on axonal delay lines terminating on coincidence detector neurons—has been experimentally confirmed in the avian nucleus laminaris (notably in barn owls by Catherine Carr and Mark Konishi), its applicability to mammals has been contested. Research by Benedikt Grothe and colleagues demonstrates that mammalian MSO neurons rely less on physical delay-line length and more on precisely timed, asymmetric glycinergic inhibition arriving via the MNTB to tune spatial receptive fields. Determining whether the mammalian system uses pure axonal delays or active synaptic inhibition remains an active area of investigation.

Hierarchical Modularity versus Early Multisensory Convergence: Classical views conceptualize the ascending auditory pathway as an isolated sensory channel that processes acoustic signals independently until reaching secondary associative cortices. However, tract-tracing and electrophysiological recordings have uncovered somatosensory and visual inputs arriving directly at early subcortical relays, including the dorsal cochlear nucleus and the inferior colliculus. This raises questions about how strictly segregated primary sensory pathways are before reaching the cerebral cortex.

Ascending versus Descending Dominance: Textbook diagrams typically emphasize the ascending, bottom-up flow of acoustic information. However, anatomical studies show that descending corticofugal and efferent fibers equal or outnumber ascending afferents at nearly every level of the pathway. The degree to which central perception represents direct environmental input versus top-down cognitive expectations remains a fundamental debate within cognitive neuroscience.

14. Related Terms & Distinctions

• Auditory Pathway vs. Auditory System: The auditory pathway refers specifically to the anatomical chain of connected neural structures and fiber tracts that transmit and process acoustic signals. The broader auditory system encompasses the pathway plus the external, middle, and inner ear structures that conduct and mechanically amplify sound waves.
• Ascending vs. Descending Auditory Pathway: The ascending pathway is the afferent conduit conveying acoustic signals from cochlear hair cells to the auditory cortex. The descending pathway is the reciprocal efferent system running from the cortex and brainstem back to the subcortical nuclei and cochlear hair cells to modulate sensitivity, tune dynamic range, and focus selective attention.
• Cochlear Nerve vs. Lateral Lemniscus: The cochlear nerve consists strictly of primary afferent axons of spiral ganglion cells bridging the cochlea and the cochlear nuclei. The lateral lemniscus is a prominent brainstem tract containing secondary, tertiary, and higher-order axons ascending from the cochlear nuclei and superior olivary complex to the midbrain inferior colliculus.
• Sensorineural Hearing Loss vs. Central Auditory Processing Disorder (CAPD): Sensorineural hearing loss involves damage to the cochlear hair cells or the cochlear nerve, reducing pure-tone hearing thresholds. Central Auditory Processing Disorder occurs when individuals exhibit normal peripheral pure-tone audiometry, but struggle to comprehend, segregate, or localize speech due to neurofunctional deficits within the brainstem or cerebral cortex.

15. Summary / Key Takeaways

• The auditory pathway is the hierarchical ascending and descending neural network responsible for converting acoustic vibrations into meaningful conscious auditory perceptions.
• Its principal anatomical stations follow an ordered sequence: cochlear hair cells → spiral ganglion (CN VIII) → cochlear nuclei → superior olivary complex → lateral lemniscus → inferior colliculus → medial geniculate body → primary and secondary auditory cortices.
• Tonotopic organization is established at the basilar membrane and preserved along every subcortical and cortical relay station, ensuring frequency-specific processing across the neuroaxis.
• Binaural sound localization begins in the superior olivary complex, where the MSO evaluates interaural time differences (ITDs) and the LSO processes interaural level differences (ILDs).
• The descending efferent system, primarily mediated through corticofugal fibers and the olivocochlear bundle, modulates peripheral cochlear sensitivity, protects against acoustic trauma, and enhances speech-in-noise perception.
• Clinical tools like the Auditory Brainstem Response (ABR) provide objective, non-invasive metrics to evaluate pathway conduction and diagnose retrocochlear pathologies such as vestibular schwannomas and auditory neuropathies.

Ultimately, the auditory pathway exemplifies the central nervous system’s capacity to preserve sub-millisecond temporal precision and fine spectral detail across successive synaptic stages. From the mechanical deflection of stereocilia to complex cortical feature extraction, this pathway transforms physical pressure waves into language, music, and spatial awareness, standing as a cornerstone of human communication and cognition.

References

• Békésy, G. von. (1960). Experiments in Hearing. McGraw-Hill.
• Grothe, B., Pecka, M., & McAlpine, D. (2010). Mechanisms of sound localization in mammals. Physiological Reviews, 90(3), 983–1012. https://doi.org/10.1152/physrev.00026.2009
• Hickok, G., & Poeppel, D. (2007). The cortical organization of speech processing. Nature Reviews Neuroscience, 8(5), 393–402. https://doi.org/10.1038/nrn2113
• Jewett, D. L., & Williston, J. S. (1971). Auditory-evoked far fields averaged from the scalp of humans. Brain, 94(4), 681–696. https://doi.org/10.1093/brain/94.4.681
• Kraus, N., & White-Schwoch, T. (2015). Unraveling the biology of auditory learning: A cognitive-sensorimotor-auditory nexus. Current Opinion in Behavioral Sciences, 4, 18–25. https://doi.org/10.1016/j.cobeha.2015.01.006
• Pickles, J. O. (2013). An Introduction to the Physiology of Hearing (4th ed.). Academic Press.
• Rauschecker, J. P., & Tian, B. (2000). Mechanisms and streams for processing of “what” and “where” in monkey auditory cortex. Proceedings of the National Academy of Sciences, 97(22), 11800–11806. https://doi.org/10.1073/pnas.97.22.11800
• Winer, J. A., & Schreiner, C. E. (Eds.). (2011). The Auditory Cortex. Springer Science & Business Media.